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Anticholinergic medications may increase Alzheimer’s risk, a new study reports

Anticholinergic medications block acetylcholine (i.e., “vagusstoff”) and inhibit parasympathetic nerve impulses by binding with this neurotransmitter’s receptors. Over 600 medications are known to trigger some degree of anticholinergic activity (Ghossein, Kang, & Lakhkar, 2020).

Anticholinergics are a common class of drugs prescribed by doctors — or purchased over-the-counter (OTC) without a prescription — for the treatment of allergies, asthma, common cold symptoms, COPD, hay fever, hypertension, overactive bladder, Parkinson’s disease as well as psychiatric disorders, depression, and a host of other ailments.

What Is Acetylcholine and Why Is It Also Called “Vagusstoff”?

Acetylcholine (ACh) was the first neurotransmitter ever discovered by scientists. In 1921, a German-born psychobiologist and pharmacologist, Otto Loewi (1873-1961), identified a substance secreted by the vagus nerve that slowed heart rate. He named this stuff “vagusstoff,” which is German for “vagus nerve substance.” (See “How Does ‘Vagusstoff’ (Vagus Nerve Substance) Calm Us Down?“)

By the mid-20th century, vagusstoff was more commonly referred to as acetylcholine. Scientists of this era also determined that ACh mediates how the autonomic nervous system’s parasympathetic branch counterbalances “fight-or-flight” stress responses.

In the 1970s, neuroscientists discovered that acetylcholine crosses the blood-brain barrier (Cornford & Oldendorf, 1975) and works differently in the central nervous system, which includes the brain and spinal cord. As a chemical messenger in the brain, acetylcholine pathways are vital to cognitive performance and memory function.

 

Read More: Drugs that block acetylcholine speed up cognitive decline – study — Health & Wellness — Sott.net